While in degenerative diseases there is an imbalance between the load and the load-bearing capacity of a joint (easily imaginable in the case of obesity), in soft tissue rheumatism it is still unclear what exactly triggers the disease. At the moment, it is assumed that genetic influences play a role – just as in inflammatory … Rheumatism: Causes and Development
Gout is a metabolic disease caused by elevated levels of uric acid (hyperuricemia) in the blood. Because an unhealthy lifestyle with poor diet and excessive alcohol consumption contributes to elevated uric acid levels, gout is considered a disease of affluence. If the disease remains untreated for a long time, the deposition of uric acid crystals … Expert Tips for Living with Gout
Before the first gout attack occurs and the disease is discovered, gout disease often exists for years. The phase in which uric acid levels continue to rise slowly but without symptoms is called the asymptomatic phase. Typical gout symptoms do not become noticeable until the level reaches a critical point and a gout attack occurs. … Typical Gout Symptoms
Just like rheumatoid arthritis, collagenoses are among the inflammatory rheumatic diseases in which the immune system produces antibodies against the body’s own components. In this case, the connective tissue is the target of the attack by the autoantibodies, which trigger chronic inflammation there. What are collagenoses? Collagenoses are a group of rare autoimmune diseases in … Collagenoses: Diseased Connective Tissue Throughout the Body
Treatment of collagenoses is carried out with the help of various drugs. But these suppress the immune system and therefore often have side effects. Below is information on therapy, prognosis, and the risk factors. What can be done about collagenosis? Drug suppression of the immune system takes the main role in the treatment of collagenosis. … Collagenoses: Therapy
“Whoever does sports gets more out of life!” – following this motto, millions of Germans do sports regularly. Because the soul and body stabilizing effect of recreational sports has long been medically proven. But wherever sports are played, there is also a risk of sports injury: over one million – mostly minor – sports injuries … Enzymes for Injuries and Osteoarthritis
The mighty Pharaoh Ramses II in Egypt suffered from it just as much as people in Palestine at the time of Jesus – medical historians are certain that ankylosing spondylitis is not a disease of civilization, but was already wreaking havoc 4,000 years ago. And it is probably no coincidence that ancient Egyptian papyrus scrolls … Ankylosing Spondylitis Symptoms
In the fight against rheumatic pain, effective painkillers are irreplaceable. But precisely these effective and soothing preparations often damage the mucous membrane of the stomach and intestines. Therefore, one cannot do without them. But you can arm yourself against the attack: with a special stomach protection therapy. NSAIDs for rheumatism Against rheumatic pain and swollen … Rheumatism: does your Stomach Need Protection?
A comprehensive clinical examination is the basis for selecting further diagnostic steps: General physical examination – including blood pressure, pulse, body weight, height; furthermore: Inspection (viewing). Skin and mucous membranes [ulcer (ulcer)?, ulcers (ulcers)?, mucosal pallor] Palpation (palpation) of the abdomen (abdomen) (tenderness?, knocking pain?, coughing pain?, defensive tension?, hernial orifices?, kidney bearing knocking pain?) … Mouth Ulcer: Examination
To prevent myocardial infarction (heart attack), attention must be paid to reducing individual risk factors. Behavioral risk factors Diet Excessive caloric intake and high-fat diet (high intake of saturated fatty acids, trans fatty acids – found especially in convenience foods, frozen foods, fast foods, snacks). Increased homocysteine due to deficiency of vitamin B6, B12 and … Myocardial Infarction (Heart Attack): Prevention
Laboratory parameters of 1st order – obligatory laboratory tests. Small blood count (alcohol consumption: MCV ↑). Fasting glucose (fasting blood glucose, fasting plasma glucose; preprandial plasma glucose; venous). HbA1c (long-term blood glucose value) Ferritin (iron stores) [ferritin ↑, in 29-50% of cases]. Triglycerides Total cholesterol and LDL/HDL ratio Liver parameters – alanine aminotransferase (ALT, GPT), … Fatty Liver (Steatosis Hepatis): Test and Diagnosis
Therapeutic targets Reduction of insulin resistance (decreased or abolished action of the hormone insulin) with decreased cardiovascular end-organ damage. Prevention of progression (progression) to nonalcoholic steatohepatitis (NASH) and/or hepatocellular carcinoma (HCC). In proven NASH, to prevent progressive fibrosis with development of cirrhosis (irreversible (non-reversible) damage to the liver and marked remodeling of liver tissue) and … Fatty Liver (Steatosis Hepatis): Drug Therapy
