Sun rays or corresponding artificial rays (solariums) accelerate the aging process of the skin. The UV-A and UV-B components of sunlight, respectively, are held responsible for skin aging caused by light.
These lead to changes in structural proteins (collagen, elastin), connective tissue cells and enzymes, which are metabolic accelerators. In addition to skin damage, pigmentary disorders and chronic degenerative changes often occur. The development of chronic dermatitis actinica (skin inflammation caused by UV radiation) is also favored. In people with light skin, tanning has the same effect as using a sunscreen (sunscreen) with a sun protection factor of 2-3, which can delay the occurrence of sunburns, but does not provide protection against mutations (permanent changes in the genetic material).
Even small doses of UV radiation lead to mutations of the skin, i.e. sunburns are not necessary for the development of skin cancer!
UV light exposure is thus a major risk factor for the development of non-melanoma skin cancer (NMSC), i.e. essentially basal cell carcinoma (BCC; basal cell carcinoma) and squamous cell carcinoma of the skin, but also leads to malignant melanoma (black skin cancer). Basal cell carcinoma and malignant melanoma are primarily associated with intermittent sun exposure and sunburns, while for squamous cell carcinoma, lifetime cumulative UV dose is significant.According to an Australian study, years of sunscreen use reduced the risk of melanoma and squamous cell carcinoma of the skin by approximately 50%, but the risk of basal cell carcinoma was not affected.
Zinc oxide nanoparticles from sunscreens probably cannot cause skin damage. In one study, no elevated zinc oxide levels beyond the stratum corneum could be detected even after multiple hourly or daily applications.
Caution. Heat, humidity and UV rays stimulate acne. Sebum production and skin exfoliation are stimulated, causing pores to clog.
