Gout (Hyperuricemia): Medical History

Medical history (history of illness) represents an important component in the diagnosis of hyperuricemia or gout. Family history Is gout common in your family? Are there any hereditary diseases in your family? Social anamnesis Current medical history/systemic medical history (somatic and psychological complaints). Do you suffer from joint pain? Which joints are affected? Is the … Gout (Hyperuricemia): Medical History

Gout (Hyperuricemia): Or something else? Differential Diagnosis

Blood, hematopoietic organs-immune system (D50-D90). Sarcoidosis (synonyms: Boeck’s disease; Schaumann-Besnier’s disease) – systemic disease of connective tissue with granuloma formation. Infectious and parasitic diseases (A00-B99). Gonorrhea (gonorrhea) – sexually transmitted infectious disease. Musculoskeletal system and connective tissue (M00-M99). Acute septic arthritis – joint inflammation caused by pathogens such as bacteria. Activated osteoarthritis – arthritis associated … Gout (Hyperuricemia): Or something else? Differential Diagnosis

Gout (Hyperuricemia): Complications

The following are the most important diseases or complications that may be contributed to by hyperuricemia or gout: Eyes and eye appendages (H00-H59). Iridopathia urica – eye involvement in gout. Endocrine, nutritional and metabolic diseases (E00-E90). Hyperlipidemia/dyslipidemia (lipid metabolism disorders). Hypercholesterolemia (LDL cholesterol elevation). HDL cholesterol decrease Hypertriglyceridemia (excessive triglyceride levels in the blood). Cardiovascular … Gout (Hyperuricemia): Complications

Gout (Hyperuricemia): Examination

A comprehensive clinical examination is the basis for selecting further diagnostic steps: General physical examination-including blood pressure, pulse, body temperature, body weight, height [overweight (obesity)]; furthermore: Inspection (viewing). Skin and mucous membranes; due tohyperuricemia (gout): Acute gout: podagra – severe joint pain in the metatarsophalangeal joint of the big toe; other joints frequently affected are … Gout (Hyperuricemia): Examination

Gout (Hyperuricemia): Drug Therapy

Goiter (goiter) change therapy goals In an acute attack of gout, the primary goal is to relieve the patient’s discomfort (analgesia) and anti-inflammation (anti-inflammation). In cases of confirmed gout disease, causal uric acid-lowering therapy should be started as soon as possible: The goal is to permanently reduce hyperuricemia and thus prevent a new gout attack … Gout (Hyperuricemia): Drug Therapy

Gout (Hyperuricemia): Therapy

General measures Gout attack: cooling, immobilization, and elevation of the extremity; administration of an analgesic/pain reliever (e.g., indomethacin). Nicotine restriction (refrain from tobacco use). Limited alcohol consumption (men: max. 25 g alcohol per day; women: max. 12 g alcohol per day) or, if necessary, abstain from alcohol. A daily alcohol consumption of 100 g leads … Gout (Hyperuricemia): Therapy

Gout (Hyperuricemia): Micronutrient Therapy

Within the framework of micronutrient medicine (vital substances), the following vital substance (micronutrient) is used for prevention and supportive therapy. Vitamin C: thereby a dosage of 500 mg/day was used. The above vital substance recommendations were created with the help of medical experts. All statements are supported by scientific studies with high levels of evidence. … Gout (Hyperuricemia): Micronutrient Therapy

Gout (Hyperuricemia): Prevention

To prevent hyperuricemia or gout, attention must be paid to reducing individual risk factors. Behavioral risk factors Diet Increased dietary purine intake, e.g., due to excessive meat consumption (especially offal) Sugar substitutes sorbitol, xylitol and fructose in high doses – The consumption of soft drinks and the associated high intake of fructose leads to an … Gout (Hyperuricemia): Prevention